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・ Toxobotys
・ Toxobotys aureans
・ Toxobotys boveyi
・ Toxobotys praestans
・ Toxocampa
・ Toxocampina
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・ Toxocara canis
・ Toxocara cati
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Toxic encephalopathy
・ Toxic epidermal necrolysis
・ Toxic equivalency factor
・ Toxic food environment
・ Toxic Girl
・ Toxic granulation
・ Toxic Grind
・ Toxic heavy metal
・ Toxic Holocaust
・ Toxic Holocaust / Inepsy
・ Toxic Holocaust / Midnight
・ Toxic Holocaust / Oprichniki
・ Toxic hotspot
・ Toxic injury
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Toxic encephalopathy : ウィキペディア英語版
Toxic encephalopathy
Toxic encephalopathy is a degenerative neurologic disorder caused by exposure to toxic substances like organic solvents. Exposure to toxic substances can lead to a variety of symptoms, characterized by an altered mental status, memory loss, and visual problems. Toxic encephalopathy can be caused by various chemicals, some of which are commonly used in everyday life. Toxic encephalopathy can permanently damage the brain and currently, treatment is mainly just for the symptoms.
== Signs and symptoms ==
"Encephalopathy" is a general term describing brain malfunctions and "toxic" asserts that the malfunction is caused by toxins on the brain. The most prominent characteristic of toxic encephalopathy is an altered mental status. Toxic encephalopathy has a wide variety of symptoms, which can include memory loss, small personality changes/increased irritability, insidious onset of concentration difficulties, involuntary movements, fatigue, seizures, arm strength problems, and depression. Neurobehavioural effects of occupational exposure to organic solvents exists among construction painters. The condition may also be referred to as substance-induced persistent dementia. Acute intoxication symptoms include lightheadedness, dizziness, headache and nausea, and regular cumulative exposure to these toxic solvents over a number of years puts the individual at high risk for developing toxic encephalopathy.
Magnetic Resonance Imaging (MRI) analyses have also demonstrated increased rates of dopamine synthesis in the putamen, reduced anterior and total corpus callosum volume, demyelination in the parietal white matter, basal ganglia, and thalamus, as well as atypical activation of frontal areas of the brain due to neural compensation. A thorough and standard diagnostic process is paramount with toxic encephalopathy, including a careful occupational history, medical history, and standardized imaging/neurobehavioral testing.

抄文引用元・出典: フリー百科事典『 ウィキペディア(Wikipedia)
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